10 Einträge von 59 mit heart anticoagulation
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Heart Failure IV A 66-year-old man is brought to the emergency department because of a 2-hour history of crushing substernal chest pain radiating to the left shoulder. His pulse is 100/min and blood pressure is 85/50 mmHg. Pulse oximetry on room air Anticoagulation B. Thrombolysis C. Inotropes D. Diuretics E. Percutaneous coronary intervention About Clinical Odyssey Why trust us Pricing For organizations Editorial Authors Peer reviewers Medical Joyworks, LLC About Jobs Contac
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Heart Failure IV A 66-year-old man is brought to the emergency department because of a 2-hour history of crushing substernal chest pain radiating to the left shoulder. His pulse is 100/min and blood pressure is 85/50 mmHg. Pulse oximetry on room air Anticoagulation B. Thrombolysis C. Inotropes D. Diuretics E. Percutaneous coronary intervention About Clinical Odyssey Why trust us Pricing For organizations Editorial Authors Peer reviewers Medical Joyworks, LLC About Jobs Contac
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heart rate drops to 80/min. She is averse to frequent blood tests. Considering the risk of thromboembolism in this patient, which of the following is the best management option? A. Warfarin B. Aspirin C. Edoxaban D. Enoxaparin E. No anticoagulation W anticoagulation Want to continue practicing? Subscribe to Clinical Odyssey today. Enjoy unlimited access to 600+ learning modules. Safely improve your skills, anytime and anywhere. Get answers to your follow-up questions from prac
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heart rate drops to 80/min. She is averse to frequent blood tests. Considering the risk of thromboembolism in this patient, which of the following is the best management option? A. Warfarin B. Aspirin C. Edoxaban D. Enoxaparin E. No anticoagulation W anticoagulation Want to continue practicing? Subscribe to Clinical Odyssey today. Enjoy unlimited access to 600+ learning modules. Safely improve your skills, anytime and anywhere. Get answers to your follow-up questions from prac
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heart failure. He had an inferior NSTEMI one year before, which was treated with percutaneous coronary intervention and placement of a stent. He was also found to have hypertension and dyslipidemia at that time. Unfortunately, he defaulted of treatme anticoagulation. Duncan responded well to your therapy. Since he was stable on rate control alone, you opted against rhythm control. Want to continue playing? Subscribe to Clinical Odyssey today. Enjoy unlimited access to 600+ lea
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heart failure. He had an inferior NSTEMI one year before, which was treated with percutaneous coronary intervention and placement of a stent. He was also found to have hypertension and dyslipidemia at that time. Unfortunately, he defaulted of treatme anticoagulation. Duncan responded well to your therapy. Since he was stable on rate control alone, you opted against rhythm control. Want to continue playing? Subscribe to Clinical Odyssey today. Enjoy unlimited access to 600+ lea
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Datum der Indexierung 07.11.2021 20:19:34
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Heart Failure IV A 66-year-old man is brought to the emergency department because of a 2-hour history of crushing substernal chest pain radiating to the left shoulder. His pulse is 100/min and blood pressure is 85/50 mmHg. Pulse oximetry on room air Anticoagulation B. Thrombolysis C. Inotropes D. Diuretics E. Percutaneous coronary intervention About Clinical Odyssey Why trust us Pricing Subscribe For organizations Editorial Authors Peer reviewers Medical Joyworks, LLC About J
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Heart Failure IV A 66-year-old man is brought to the emergency department because of a 2-hour history of crushing substernal chest pain radiating to the left shoulder. His pulse is 100/min and blood pressure is 85/50 mmHg. Pulse oximetry on room air Anticoagulation B. Thrombolysis C. Inotropes D. Diuretics E. Percutaneous coronary intervention About Clinical Odyssey Why trust us Pricing Subscribe For organizations Editorial Authors Peer reviewers Medical Joyworks, LLC About J
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Heart Failure IV A 66-year-old man is brought to the emergency department because of a 2-hour history of crushing substernal chest pain radiating to the left shoulder. His pulse is 100/min and blood pressure is 85/50 mmHg. Pulse oximetry on room air Anticoagulation B. Thrombolysis C. Inotropes D. Diuretics E. Percutaneous coronary intervention Want to continue practicing? Subscribe to Clinical Odyssey today. Enjoy unlimited access to 600+ learning modules. Safely improve your
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Heart Failure IV A 66-year-old man is brought to the emergency department because of a 2-hour history of crushing substernal chest pain radiating to the left shoulder. His pulse is 100/min and blood pressure is 85/50 mmHg. Pulse oximetry on room air Anticoagulation B. Thrombolysis C. Inotropes D. Diuretics E. Percutaneous coronary intervention Want to continue practicing? Subscribe to Clinical Odyssey today. Enjoy unlimited access to 600+ learning modules. Safely improve your
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Datum der Indexierung 11.03.2022 22:49:54
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heart chambers due to increased intrapericardial pressure; this can be due to the accumulation of blood, pus, effusion (transudate or exudate) or air within the pericardial space. The hallmark of cardiac tamponade is the rapid rise and equalization o anticoagulation and left atrial access—are the most common cause of acute cardiac tamponade. Dyspnea Dyspnea is the most common symptom. It is caused by pulmonary congestion due to reduced cardiac output and impaired systemic veno
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heart chambers due to increased intrapericardial pressure; this can be due to the accumulation of blood, pus, effusion (transudate or exudate) or air within the pericardial space. The hallmark of cardiac tamponade is the rapid rise and equalization o anticoagulation and left atrial access—are the most common cause of acute cardiac tamponade. Dyspnea Dyspnea is the most common symptom. It is caused by pulmonary congestion due to reduced cardiac output and impaired systemic veno
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heart chambers due to increased intrapericardial pressure; this can be due to the accumulation of blood, pus, effusion (transudate or exudate) or air within the pericardial space. The hallmark of cardiac tamponade is the rapid rise and equalization o anticoagulation and left atrial access—are the most common cause of acute cardiac tamponade. Dyspnea Dyspnea is the most common symptom. It is caused by pulmonary congestion due to reduced cardiac output and impaired systemic veno
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heart chambers due to increased intrapericardial pressure; this can be due to the accumulation of blood, pus, effusion (transudate or exudate) or air within the pericardial space. The hallmark of cardiac tamponade is the rapid rise and equalization o anticoagulation and left atrial access—are the most common cause of acute cardiac tamponade. Dyspnea Dyspnea is the most common symptom. It is caused by pulmonary congestion due to reduced cardiac output and impaired systemic veno
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heart disease and atrial fibrillation, a transient ischemic attack (TIA) is a strong possibility; the subtherapeutic INR (indicating inadequate anticoagulation) lends further credence to this suspicion.\n\nOther differentials include hypoglycemia, el anticoagulation) lends further credence to this suspicion.\n\nOther differentials include hypoglycemia, electrolyte disturbances, a subdural hematoma or a cerebral tumor (with hemorrhage inside); however, these are made unlikely b
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heart disease and atrial fibrillation, a transient ischemic attack (TIA) is a strong possibility; the subtherapeutic INR (indicating inadequate anticoagulation) lends further credence to this suspicion.\n\nOther differentials include hypoglycemia, el anticoagulation) lends further credence to this suspicion.\n\nOther differentials include hypoglycemia, electrolyte disturbances, a subdural hematoma or a cerebral tumor (with hemorrhage inside); however, these are made unlikely b
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heart attack’. He was then administered Aspirin and sublingual Nitroglycerin and rushed to hospital. Now, at presentation, his chest pain has subsided completely. His medical history is unremarkable, while a full blood count and chest x-ray are norma Anticoagulation clear 3. Calcium Channel Blockers check 4. Long-acting Nitrates check
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heart attack’. He was then administered Aspirin and sublingual Nitroglycerin and rushed to hospital. Now, at presentation, his chest pain has subsided completely. His medical history is unremarkable, while a full blood count and chest x-ray are norma Anticoagulation clear 3. Calcium Channel Blockers check 4. Long-acting Nitrates check
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Heart Failure IV A year old man is brought to the emergency department because of a hour history of crushing substernal chest pain radiating to the left shoulder His pulse is min and blood pressure is mmHg Pulse oximetry on room air Anticoagulation B Thrombolysis C Inotropes D
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heart failure or myocardial dysfunction - Diffuse coronary artery ectasia and aneurysm formation - Myocardial infarction - Myocarditis - Valvulitis, most often of the mitral valve - Pericarditis and pericardial effusions - Systemic arterial aneurysm anticoagulation should avoid contact or high-impact sports indefinitely, again because of the risk of bleeding. In addition, the level of physical activity in patients at high risk for future myocardial infarction (i.e. risk level
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heart failure or myocardial dysfunction - Diffuse coronary artery ectasia and aneurysm formation - Myocardial infarction - Myocarditis - Valvulitis, most often of the mitral valve - Pericarditis and pericardial effusions - Systemic arterial aneurysm anticoagulation should avoid contact or high-impact sports indefinitely, again because of the risk of bleeding. In addition, the level of physical activity in patients at high risk for future myocardial infarction (i.e. risk level
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heart attack’ He was then administered Aspirin and sublingual Nitroglycerin and rushed to hospital Now at presentation his chest pain has subsided completely His medical history is unremarkable while a full blood count and chest x ray are norma Anticoagulation clear Calcium Channel Blocke
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